DSpace Community:https://digitallibrary.bldedu.ac.in/xmlui/handle/123456789/47322026-04-17T17:09:51Z2026-04-17T17:09:51ZSpontaneous occurrence and expulsion of a massive Steinstrasse.Kundargi, VinayPatil, SantoshShukla, VikasPatil, Siddanagouda Bhttps://digitallibrary.bldedu.ac.in/xmlui/handle/123456789/54752024-03-12T05:14:59Z2024-01-01T00:00:00ZTitle: Spontaneous occurrence and expulsion of a massive Steinstrasse.
Authors: Kundargi, Vinay; Patil, Santosh; Shukla, Vikas; Patil, Siddanagouda B2024-01-01T00:00:00ZA Rare Case of Nicolau’s Syndrome (Embolia Cutis Medicamentosa) Following Intramuscular Diclofenac Sodium Injection in a Young AdultM. S. Kotennavar, Aravind V. PatilPradeep Jaju, Narendra Ballalhttps://digitallibrary.bldedu.ac.in/xmlui/handle/123456789/54662024-03-08T07:19:24Z2024-01-01T00:00:00ZTitle: A Rare Case of Nicolau’s Syndrome (Embolia Cutis Medicamentosa) Following Intramuscular Diclofenac Sodium Injection in a Young Adult
Authors: M. S. Kotennavar, Aravind V. Patil; Pradeep Jaju, Narendra Ballal
Abstract: Nicolau syndrome (embolia cutis medicamentosa) is a condition where we see variable degrees of tissue necrosis including the skin and deeper tissues, due to an iatrogenic cause, intramuscular, subcutaneous, intra-articular injections, could be some of them. It occurs due to intravascular inoculation leading to crystal embolization causing thrombotic occlusion, vasospasm, and marked inflammation and surrounding necrosis. Here, we present a case of a 35-year-old gentleman who presented to us with bluish-purple discoloration of the skin followed by a necrotic patch and ulceration with multiple hyperpigmented spots over the back and lower limb post intramuscular diclofenac sodium injection. Routine investigation showed neutrophilia and thrombocytosis with increased creatinine. A diagnosis of Nicolau’s syndrome (embolia cutis medicamentosa) was made clinically, which was confirmed by histopathology following biopsy. Adequate surgical debridement and a combination therapy of analgesics, intravenous antibiotics, intravenous anticoagulants, and vasoactive therapy were administered. Split thickness skin grafting was done once the wound was healthy with rich granulation tissue, after 4 weeks of index surgery in this case. Patient recovered uneventfully.2024-01-01T00:00:00ZPort-wine stain associated with membranous aplasia cutis congenita and hair collar signInamadar, Arunhttps://digitallibrary.bldedu.ac.in/xmlui/handle/123456789/54572024-03-07T03:33:33Z2024-01-01T00:00:00ZTitle: Port-wine stain associated with membranous aplasia cutis congenita and hair collar sign
Authors: Inamadar, Arun2024-01-01T00:00:00ZCoexistence of annular polycyclic, morpheaform and atrophic lesions in neonatal lupus erythematosusKulkarni, ShrutiAdya, Keshavmurthy AJanagond, Ajit BInamadar, Arunhttps://digitallibrary.bldedu.ac.in/xmlui/handle/123456789/48002023-04-19T11:02:03Z2022-01-01T00:00:00ZTitle: Coexistence of annular polycyclic, morpheaform and atrophic lesions in neonatal lupus erythematosus
Authors: Kulkarni, Shruti; Adya, Keshavmurthy A; Janagond, Ajit B; Inamadar, Arun
Abstract: Neonatal lupus erythematosus (NLE) occurs due
to transplacental transfer of autoantibodies in
newborns of mothers with clinical or subclinical
collagen vascular diseases. Anti-Ro/SSA antibodies
are strongly associated with NLE. Anti-La/SSB and
anti-U1
-RNP antibodies are less frequent. Cutaneous and cardiac manifestations are prominent
of NLE. Nearly half of the cases show either cutaneous or cardiac features, and 10% show both. Skin
lesions may be congenital or develop within 12–16
weeks postpartum2022-01-01T00:00:00Z