<?xml version="1.0" encoding="UTF-8"?>
<rdf:RDF xmlns:rdf="http://www.w3.org/1999/02/22-rdf-syntax-ns#" xmlns="http://purl.org/rss/1.0/" xmlns:dc="http://purl.org/dc/elements/1.1/">
  <channel rdf:about="https://digitallibrary.bldedu.ac.in/xmlui/handle/123456789/4733">
    <title>DSpace Collection:</title>
    <link>https://digitallibrary.bldedu.ac.in/xmlui/handle/123456789/4733</link>
    <description />
    <items>
      <rdf:Seq>
        <rdf:li rdf:resource="https://digitallibrary.bldedu.ac.in/xmlui/handle/123456789/6327" />
        <rdf:li rdf:resource="https://digitallibrary.bldedu.ac.in/xmlui/handle/123456789/6326" />
        <rdf:li rdf:resource="https://digitallibrary.bldedu.ac.in/xmlui/handle/123456789/5475" />
        <rdf:li rdf:resource="https://digitallibrary.bldedu.ac.in/xmlui/handle/123456789/5466" />
      </rdf:Seq>
    </items>
    <dc:date>2026-08-05T14:51:33Z</dc:date>
  </channel>
  <item rdf:about="https://digitallibrary.bldedu.ac.in/xmlui/handle/123456789/6327">
    <title>Management of traumatic tractional corectopia</title>
    <link>https://digitallibrary.bldedu.ac.in/xmlui/handle/123456789/6327</link>
    <description>Title: Management of traumatic tractional corectopia
Authors: Keerti Wali, Vaishnavi Patil, Vallabha K
Abstract: Corectopia is the displacement of the pupil. It can be&#xD;
isolated congenital anamoly or associated with syndromes&#xD;
like Ectopia lentis et pupillae or Axenfeld–Reiger anamoly.&#xD;
[1] Aquired corectopia is often caused by trauma, surgery,&#xD;
or anterior chamber inflammation.[2] It is caused by a&#xD;
fibrous band exerting traction on the iris and may be&#xD;
either stationary or progressive.[2] Treatment is tailored&#xD;
considering the site of traction and associated comorbidities.&#xD;
We present a case of traumatic tractional corectopia and its&#xD;
management.</description>
    <dc:date>2026-02-01T00:00:00Z</dc:date>
  </item>
  <item rdf:about="https://digitallibrary.bldedu.ac.in/xmlui/handle/123456789/6326">
    <title>Adult Adenoid Hypertrophy Mimicking a Nasopharyngeal Mass: A Diagnostic Challenge</title>
    <link>https://digitallibrary.bldedu.ac.in/xmlui/handle/123456789/6326</link>
    <description>Title: Adult Adenoid Hypertrophy Mimicking a Nasopharyngeal Mass: A Diagnostic Challenge
Authors: Soumya Kori, Rupali Kumari , Savitri M. Nerune
Abstract: The adenoids, also known as the nasopharyngeal tonsils, come from lymphoid tissue in the pharyngeal&#xD;
mucosa. They are part of Waldeyer’s ring, which is a circular arrangement of lymphoid tissue. This ring&#xD;
includes the pharyngeal (adenoid), palatine, lingual, and tubal tonsils. These tonsils are located at the&#xD;
entrance of the aerodigestive tract.&#xD;
They play an important role in mucosal immunity during early life by trapping inhaled pathogens and helping&#xD;
with antigen presentation. This contributes to both humoral and cell-mediated immune responses.&#xD;
Adenoid hypertrophy is predominantly a pediatric condition and rarely persists into adulthood due to&#xD;
physiological involution after puberty. When present in adults, it may clinically and radiologically mimic&#xD;
various nasopharyngeal pathologies, including malignancy.&#xD;
We report a case of a 51-year-old woman presenting with long-standing left-sided nasal obstruction. She also&#xD;
presented with snoring, mouth breathing and headache. Clinical examination revealed a deviated nasal</description>
    <dc:date>2026-03-01T00:00:00Z</dc:date>
  </item>
  <item rdf:about="https://digitallibrary.bldedu.ac.in/xmlui/handle/123456789/5475">
    <title>Spontaneous occurrence and expulsion of a massive Steinstrasse.</title>
    <link>https://digitallibrary.bldedu.ac.in/xmlui/handle/123456789/5475</link>
    <description>Title: Spontaneous occurrence and expulsion of a massive Steinstrasse.
Authors: Kundargi, Vinay; Patil, Santosh; Shukla, Vikas; Patil, Siddanagouda B</description>
    <dc:date>2024-01-01T00:00:00Z</dc:date>
  </item>
  <item rdf:about="https://digitallibrary.bldedu.ac.in/xmlui/handle/123456789/5466">
    <title>A Rare Case of Nicolau’s Syndrome (Embolia Cutis Medicamentosa) Following Intramuscular Diclofenac Sodium Injection in a Young Adult</title>
    <link>https://digitallibrary.bldedu.ac.in/xmlui/handle/123456789/5466</link>
    <description>Title: A Rare Case of Nicolau’s Syndrome (Embolia Cutis Medicamentosa) Following Intramuscular Diclofenac Sodium Injection in a Young Adult
Authors: M. S. Kotennavar, Aravind V. Patil; Pradeep Jaju, Narendra Ballal
Abstract: Nicolau syndrome (embolia cutis medicamentosa) is a condition where we see variable degrees of tissue necrosis including the skin and deeper tissues, due to an iatrogenic cause, intramuscular, subcutaneous, intra-articular injections, could be some of them. It occurs due to intravascular inoculation leading to crystal embolization causing thrombotic occlusion, vasospasm, and marked inflammation and surrounding necrosis. Here, we present a case of a 35-year-old gentleman who presented to us with bluish-purple discoloration of the skin followed by a necrotic patch and ulceration with multiple hyperpigmented spots over the back and lower limb post intramuscular diclofenac sodium injection. Routine investigation showed neutrophilia and thrombocytosis with increased creatinine. A diagnosis of Nicolau’s syndrome (embolia cutis medicamentosa) was made clinically, which was confirmed by histopathology following biopsy. Adequate surgical debridement and a combination therapy of analgesics, intravenous antibiotics, intravenous anticoagulants, and vasoactive therapy were administered. Split thickness skin grafting was done once the wound was healthy with rich granulation tissue, after 4 weeks of index surgery in this case. Patient recovered uneventfully.</description>
    <dc:date>2024-01-01T00:00:00Z</dc:date>
  </item>
</rdf:RDF>

