Please use this identifier to cite or link to this item: http://20.193.157.4:9595/xmlui/handle/123456789/1261
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dc.contributor.authorKusal K Das, Jargar JG Sikha Saha Sayeed Yendigiri Sashi Bala singh-
dc.date.accessioned2019-11-19T06:39:05Z-
dc.date.available2019-11-19T06:39:05Z-
dc.date.issued2015-
dc.identifier.issn02537613-
dc.identifier.urihttp://hdl.handle.net/123456789/1261-
dc.description.abstractLead (Pb) is a longknown poison of environment and industrial origin. Its prolonged exposure affects cellular material and alters cellular genetics and produces oxidative damages. In this study, we investigated the exposure of chronic sustained hypoxia or lead acetate alone or in combination with or without supplementation of αtocopherol on hepatic oxidative and nitrosative stress in ratsen_US
dc.language.isoenen_US
dc.publisherBLDE(Deemed to be University)en_US
dc.subjectαtocopherol, hypoxia, lead acetate, nitrosative stress, oxidative stressen_US
dc.titleAlpha tocopherol prevents lead acetate and hypoxia induced hepatic dysfunction.en_US
dc.typeArticleen_US
Appears in Collections:Faculty of Physiology

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