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| DC Field | Value | Language |
|---|---|---|
| dc.contributor.author | Kulkarni, Shruti | - |
| dc.contributor.author | Adya, Keshavmurthy A | - |
| dc.contributor.author | Janagond, Ajit B | - |
| dc.contributor.author | Inamadar, Arun | - |
| dc.date.accessioned | 2023-04-19T11:02:03Z | - |
| dc.date.available | 2023-04-19T11:02:03Z | - |
| dc.date.issued | 2022 | - |
| dc.identifier.uri | http://hdl.handle.net/123456789/4800 | - |
| dc.description.abstract | Neonatal lupus erythematosus (NLE) occurs due to transplacental transfer of autoantibodies in newborns of mothers with clinical or subclinical collagen vascular diseases. Anti-Ro/SSA antibodies are strongly associated with NLE. Anti-La/SSB and anti-U1 -RNP antibodies are less frequent. Cutaneous and cardiac manifestations are prominent of NLE. Nearly half of the cases show either cutaneous or cardiac features, and 10% show both. Skin lesions may be congenital or develop within 12–16 weeks postpartum | en_US |
| dc.language.iso | en | en_US |
| dc.publisher | BLDE(DU) | en_US |
| dc.subject | polycyclic | en_US |
| dc.subject | neonatal lupus | en_US |
| dc.title | Coexistence of annular polycyclic, morpheaform and atrophic lesions in neonatal lupus erythematosus | en_US |
| dc.type | Article | en_US |
| Appears in Collections: | BMJ Case Reports | |
Files in This Item:
| File | Description | Size | Format | |
|---|---|---|---|---|
| e252434.full.pdf | 556.36 kB | Adobe PDF |  View/Open |
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